Kuyaziwa ukuthi anti-depressants angaba nemiphumela yocansi. Zingaholela ezinkingeni ngesifiso, ukuvusa, ukujula, ne-orgasm. Lezi zinkinga zobulili zihlotshaniswa nokusetshenziswa kwezigaba ezimbili zezidakamizwa-i-serotonin reuptake inhibitors (SSRIs) kanye ne-serotonin-norepinephrine reuptake inhibitors (SNRI).
Ekuqaleni kakhulu ekuthuthukiseni kwama-SSRIs, kwafunyanwa ukuthi le mithi ingabangela ubunzima ngokujula.
Eqinisweni, le mithi empeleni iqondiswe ngamadoda anezinkinga nge- ejaculation ngaphambi kwesikhathi ! Kungani izidakamizwa zokudambisa izidakamizwa zingaba nemiphumela emibi kakhulu empilweni yezocansi, ikakhulukazi kubantu? Akunandaba nhlobo nemizwelo. Esikhundleni salokho, kuhlobene nendima ye-serotonin namanye ama-neurotransmitter ekuvusweni nasekuseni.
I-Serotonin ne-Ejaculation
Ubulili, ngezindlela eziningi, umsebenzi wengqondo. Ukuvuswa komzimba kungenziwa ngokugqugquzela ngokuqondile. Kungabuye kwenzeke ngaphandle kokuvuselela okuqondile. Abacwaningi bezocansi bavame ukuphakamisa "imicabango enhle" njengokungenelela, ngoba ukucabanga ngocansi kungaba ukuvukela ngaphakathi. Lokho kusebenza kanjani? Indlela efanayo imicabango eminingi esetshenziselwa ngayo-ngokukhiqizwa kwama-neurotransmitter ahlukahlukene. Yileyo ndlela amangqamuzana omzimba axhuma ngayo. Bakhulula futhi baphendule ezithombeni ze-neurotransmitters. Ama-neurotransmitter anjalo afaka i-serotonin, i-norepinephrine, ne-dopamine.
Ukuqonda okuningi kwendima yama-neurotransmitters kanye nezinhlaka zobuchopho ekuvusweni nase-orgasm kuvela ekucwaningweni kwezilwane. Kodwa-ke, kuye kwaba nezifundo zabantu. Ngokucwaninga, ososayensi baye bafunda ukuthi kunezindawo eziningana zobuchopho ezihlobene nomsebenzi wesilisa ocansini. Ngokuyinhloko, indawo yangaphambili ye-hypothalamus ibaluleke kakhulu.
I-MPOA, njengoba kuthiwa ngezinye izikhathi, yilapho isenzo socansi ezivela ezingxenyeni ezihlukahlukene zomzimba zihlangana ndawonye ukuze kusetshenzwe. Kubuye kube yingxenye yobuchopho obuqala izinhlobo eziningana zokuphendula ngezocansi. Ubuchopho buyathumela izibonakaliso phansi kwentambo yomgogodla obangela umzimba ukuba uvuke kuqala bese u-orgasm. Izingxenye ze-amygdala kanye ne-cortex ye-parietal nazo zibalulekile ekulawulweni kwe-ejaculation.
I-neurotransmitters yindlela yokwenza lezi zimpawu. Ukuhlolwa kwamagundane kukhombise ukuthi kungenzeka ukuthi kubangele ama-rats ukuba asebenze nge-orgasm nje ngokuvuselela ezinye izimpendulo ze-neurotransmitter ebuchosheni. (Abamukelayo yizindawo lapho izibopho ze-neurotransmitter zibopha khona. Kunezinhlobo eziningi ze-receptors ezingaphendula kumuntu ngamunye, noma inhlanganisela, yezokudlulisa ezihlukahlukene. Akuzona zonke izinhlobo ezisatshalaliswa ngokufanayo ngobuchopho.)
Kubantu, i-serotonin yi-neurotransmitter ehlotshaniswa ngokucacile ne-ejaculation. Ama-SSRI asebenza ngokuvimbela amaseli ukuthi angabuyiselwa kabusha i-serotonin. Lokhu kusho ukuthi izimpawu ezibangelwa i-serotonin zandiswa, zihlala isikhathi eside. Ukusetshenziswa okungapheli kwama-SSRIs kuye kwaboniswa ukwandisa isikhathi phakathi kokuklanywa nokukhishwa kwamanzi emadodeni. Yingakho inqunywe njengendlela yokwelapha i-ejaculation ngaphambi kwesikhathi.
Ngokuthakazelisayo, ukuhlolwa kwamagundane kubonise ukuthi lapho i-serotonin ishintsha imiphumela yayo. Uma i-serotonin ingenwa kwezinye izingxenye zobuchopho besilwane, kubangele ukubambezeleka ekujuleni. Kwezinye izindawo zobuchopho, kubangela ukuba ukuqhuma kuqhume.
I-dopamine nayo idlala indima ekwenzeni i-ejaculation, nakuba indima yayo ingazange ihlolwe ngokujulile njengayo ye-serotonin. Ukuhlolwa kwamagundane kubonisa ukuthi ukugqugquzela i-dopamine kungabangela ukuba ukuqhutshwa kwe-ejaculation kwenzeke. Kubantu, kukhona ucwaningo lokusekela lokhu. Ama-schizophrenics aphethwe izidakamizwa ezilwa no-psychotic ezivimbela uhlobo oluthile lwe-dopamine receptor (i-D2-like receptors) cishe zithola kunzima, noma akunakwenzeka, ukuba ku-ejaculate.
Lezo zidakamizwa ziye zahlolelwa kumadoda ane-ejaculation ngaphambi kwesikhathi. Ngokufanayo nama-SSRIs, kubonakala sengathi banweba isikhathi phakathi kwe-arousal ne-ejaculation. Ngaphezu kwalokho, kunomthamo omncane wedatha obonisa ukuthi ukuguqulwa kwamagciwane e-dopamine receptors kungenza amanye amadoda abe nakho ukuzwa i-ejaculation ngaphambi kwesikhathi.
Ukuqonda ukujula
Ukujula ukuchazwa kuthiwa yi-propulsion yamagciwane avela emzimbeni. Kwenzeka ngezigaba ezimbili. Isigaba sokuqala ukuphuma. Yilokho lapho izingxenye ezihlukahlukene ze-semen, kuhlanganise nesidoda, zifihliwe ezigulini ezihlukahlukene kanye nezitho. Isigaba sesibili ukuxoshwa. Yilapho ukuphazamiseka okukhulu kwemisipha engxenyeni yesisu kubangela ukuba umuthi uphonswe ngaphandle kwepenisi.
Inothi, amadoda athile abenezinhlobo ezithile zokuhlinzwa komdlavuza we-prostate angahle athole "u-dry" ejaculation. Lokhu kungenxa yokuthi imizimba yabo ayisekho okwakhiqiza izingxenye zamanzi zomzimba. Cishe ingxenye eyodwa kwezintathu zomkhuhlane we-seminal uvela ku-prostate. Ezinye izigulane ezithinta ukukhiqizwa kwamanzi omzimba zingase zithinteke ngokuhlinzwa kwama-cancer.
Emadodeni, igama elithi orgasm livame ukusetshenziselwa njengendlela efanelana ngayo nokujula. Nokho, ukujula kanye ne-orgasm akuyona into efanayo. Nakuba kumadoda amaningi, i-orgasm ngokuyinhloko iyenzeka ngesikhathi sokujula, lokho akuyona into njalo. Amanye amadoda anama-multi-orgasmic angase abe nama-orgasms amaningi nge-ejaculation eyodwa kuphela. Amanye amadoda awakwazi ukukhipha i-ejaculate nhlobo. Lesi simo siyaziwa ngokuthi singenayo.
Ukuhlukanisa ukuHlelwa nokuHlelwa kwe-Ejaculation
Kubalulekile ukuthi uqaphele ukuthi ukulungiswa akudingeki ukuba u-ejaculation kwenzeke. Ukujula ngaphandle kokulungiswa akuvamile, kodwa kungenzeka. Isibonelo esivame kakhulu salokhu senzeke kubafana abasha ngenkathi belele-okuthiwa "ukukhishwa kwesikhombisa" noma "amaphupho anamanzi." Ukukhishwa kwelanga kungenziwa noma ngaphandle kokuba khona kwe-erection. Ukujula ngaphandle kokumiswa kungahle kukhuthazwe nokusebenzisa ukudlidliza kwe-penis kumadoda anezinhlobo ezithile zokulimala komgogodla. Le nqubo ibuye isetshenziswe ngezinye izikhathi ukuqoqa umuthi ngezinqubo zokusiza zokukhiqiza.
Ngokumangazayo, izindlela zezinzwa nezindlela zokuphila eziholela ekwakhiweni nasekukhuleni zihlobene, kodwa azifani. Yingakho ama-inhibitors we-PDE-5 asetshenziselwa ukusiza amadoda ane-erectile ukungasebenzi awenzi nomphumela omkhulu ekukhoneni kwabo. Zithinta ukugeleza kwegazi ngaphakathi nangaphandle kwepeni. Awukwenzi ukukhululwa kwezingxenye zezinsikazi noma ukuphazamiseka kwemisipha yokuxoshwa.
> Imithombo:
> Clement P, Giuliano F. Physiology and Pharmacology of Ejaculation. Basic Clinic Pharmacol Toxicol. 2016 Okthoba; 119 Uku-3: 18-25. i-doi: 10.1111 / bcpt.12546.
> I-Courtois F, i-Carrier S, i-Charvier K, i-Guertin PA, i-Journel NM. Ukulawulwa kwezimpendulo zesilisa zocansi. I-Curr Pharm Des. 2013; 19 (24): 4341-56.
> Giuliano F. Neurophysiology yokwakhiwa kanye nokujula. I-J Med Med. 2011 Oct; 8 Ukusekela 4: 310-5. i-doi: 10.1111 / j.1743-6109.2011.02450.x.
> Safarinejad MR. Ubudlelwano phakathi kwe-ejaculation engakafiki ngaphambi kwesikhathi kanye nama-polymorphisms ezakhiwe zofuzo lwe-dopamine transporter (SLC6A3). I-BJU Int. 2011 Jul; 108 (2): 292-6. i-doi: 10.1111 / j.1464-410X.2010.09809.x.