, Nakuba izixhumanisi phakathi kwesifo se-celiac nokuhlukunyezwa kwabesifazane zihlelwe kahle, kube nokucwaninga okuncane kakhulu kunoma yikuphi ukuxhumana phakathi kwesifo se-celiac nokusabalalisa besilisa.
Kusukela ocwaningweni olude olwenziwe, kubonakala sengathi kungase kutholakale isixhumanisi phakathi kwesifo se-celiac nokusabalalisa besilisa - njengabesifazane abanesimo, abantu abathola izifo ze-celiac ezingatholakali babonakala behlushwa ukungazali ngokweqile kunamanye amadoda.
Kodwa-ke, akuwona wonke ucwaningo olubonise isixhumanisi esinjalo, ngakho-ke ucwaningo olwengeziwe luyadingeka ngaphambi kokuba odokotela bakwazi ukusho ngokucacile ukuthi isifo se-celiac sinciphisa ukuzala kwesilisa nokuthi ngabe ukudla okungekho gluten kungasiza.
Izifo ze-Celiac kanye nokuCwaninga kwe-Malefactless
Amadoda anezifo ze- celiac ezingatholakali abonakala anezilinganiso eziphakeme kakhulu zesidoda esingavamile, kanye namazinga ajwayelekile angama-hormone.
Ngokukodwa, isifundo esisodwa sathola ukuthi amadoda angaphezu kuka-19% wabesifazane abashadile abashadile babe nemishado engaphelele, futhi ukuhlaziywa kwesilisa kuthole izinkinga nge-morphology yabo yesisu nomoya noma isakhiwo sensimu kanye nokukwazi ukuhamba.
Eqinisweni, ukukhulelwa kwesisu kwanciphisa "ngokuphawulekayo" kokubili kwamakholi amathathu anemishado engapheli, isifundo sitholile.
Ngaphezu kwalokho, esinye isifundo sathola ukuthi amadoda ane-celiac isifo esingahlosiwe ajwayele ukubhekana nokuphikiswa kwe-androgen, okusho ukuthi izidumbu zabo azange zisabele kahle kwi-hormone testosterone.
Abacwaningi bacabanga ukuthi ukumelana ne-androgen kubonakala ukuphazanyiswa okuvamile kwezinhlelo zamadoda eziphethwe yi-endocrine ezibangelwa yisifo se-celiac.
Kodwa-ke, isifundo esikhulu esivela eSweden, eshicilelwe ngo-2011, sabheka amadoda angu-7 121 athole ukuthi unezifo ezicatshangelwayo futhi wawalandela lapho esekhulile futhi ephakathi.
Lolu cwaningo lathola ukuthi amadoda atholakala ukuthi anezifo ze-celiac ayenamanani afanayo wezingane uma kuqhathaniswa nabantu ababengenayo isifo se-celiac. Kwaphetha ngokuthi amadoda asevele ebonwe ukuthi unezifo ezingenayo i-celiac ayinayo izinga eliphansi lokuzala kunelabantu abaningi.
Ukungafani Kwesilisa Nesidlo Sama-Gluten
Ucwaningo lwezifo ze-celiac kanye nokungasebenzi kwamadoda kuye kwathola ukuthi izidalwa zesilisa zathuthuka lapho amadoda abathintekayo abhekwa ukuthi banezifo ze-celiac futhi athola ukudla okungenalutho kwe-gluten. Baphinde bathola ukuthi ama-hormone amazinga abuyela emuva evamile ngemuva kokuba amadoda aqale ukudla okungenamsoco we-gluten.
Ngakho-ke, kungenzeka ukuthi amadoda angama-celiac ngaphambili ayengakwazi ukuzala uma eqala ukudla okungenayo i-gluten, okuvame ukukwenzeka nabesifazane abangama-celiac.
Noma kunjalo, akuzange kube nokucwaninga okuningi ukubonisa lokhu kuyiqiniso. Kungenzeka ukuthi ukuzala akuzange kuhlupheke emadodeni avela ekutadisheni kwesiSweden ka-2011 ngoba bebalandela ukudla okungekho gluten. Kodwa njengoba isifundo singasethiwe ukuze siphendule lo mbuzo, ayikho indlela yokwazi ukuthi ngabe lokhu kwakuyisici.
Iningi lezocwaningo mayelana nokuzala emadodeni angama-celiac zenziwa ngamashumi ama-1970 nama-80, okukhombisa isidingo esikhulu esingafinyeleleki solwazi olusha.
Ukuvivinya Izifo Ezibucayi Emadodeni Angenalutho
Ngakho-ke, uma ungumuntu ongenalwazi olungenakuchazwa, kufanele yini uhlolwe isifo se-celiac? Abanye odokotela bathi yebo, ikakhulukazi uma unezinye izimpawu zesifo se-celiac .
Kodwa-ke, abantu abaningi abahlolwayo abanesifo se-celiac banezimpawu ezimbalwa noma ngisho nezezibonakaliso ezicacile, ngakho-ke akufanele neze uthembele ezimpawu zakho ukucacisa ingozi yakho.
Imithombo:
Farthing, MJ et al. Umsebenzi we-Gonadal wesilisa ku-Celiac Disease 1. Ukungasebenzi kahle ngokobulili, ukungenzi lutho, kanye nekhwalithi yesimiso. Gut 1982; 23: 608-614.
Freeman, HJ Izinguquko Zokukhiqiza Ezihambisana Nezifo Ezibucayi. I-World Journal ye-Gastroenterology 2010; 16 (46): 5810-5814.
UZugna D et al. Izifo ze-Celiac Akuyona Isici Sengozi Yokungafaki Amadoda. Ukuzala nokuhlunga. 2011, 95 (5): 1709-13.e1-3.