Ayikho Ukwelashwa Kwezinga eliphezulu le-CRP namazinga e-Fibrinogen
Ukuhlolwa kwegazi ezimbili kuye kwagqugquzelwa njengezibikezelo zesifo senhliziyo. Zombili lezi zivivinyo zegazi- Iprotheyini esebenzayo ye-C (i-CRP) ne- fibrinogen- manje ibhekene nengozi enkulu kakhulu yokuhlasela kwenhliziyo esikhathini esizayo. Inkinga, ngokungafani nezinye izici eziyingozi (ezifana nokukhuluphala ngokweqile, ukubhema, ne-cholesterol) akucaci ukuthi kufanele kwenziwe kanjani ngezinga eliphezulu le-CRP namazinga e-fibrinogen.
I-CRP ne-Fibrinogen
I-CRP yiprotheni ekhishwe engxenyeni yegazi noma kunini lapho kukhona ukuvuvukala okusebenza emzimbeni. (Ukuvuvukala kwenzeka ekuphenduleni ukutheleleka, ukulimala, noma izimo ezihlukahlukene ezifana ne- arthritis .) Ubufakazi bubonisa ukuthi i-atherosclerosis (i- coronary artery disease ) inqubo yokuvuvukala. Abanye baze bacabange ukuthi isifo se-artery coronary singakhuthazwa yi-infection. Iqiniso eliphakamisa amazinga e-CRP ahlotshaniswa nengozi eyengeziwe yokwehla kwenhliziyo ithambekele ekusekeleni ubudlelwane obuhlongozwayo phakathi kokuvuvukala kanye ne-atherosclerosis.
I-Fibrinogen yisici sokuvala igazi. Amaphutha amaningi kakhulu e-myocardial (ukuhlaselwa kwenhliziyo) manje ayaziwa ngenxa ye-thrombosis ephawulekayo, noma ukwakheka okungazelelwe kwe-clot yegazi esakhiweni se-plaque ye-atherosclerotic. Ngakho-ke, kunengqondo ukuthi ukuphakama kwamazinga e-fibrinogen (okungukuthi, amaprotheni akhuthaza ukugaya igazi) kuzohambisana nenengozi yokwanda kwenhliziyo.
Ingabe amazinga aphezulu e-CRP nama-Fibrinogen angaphathwa?
Impendulo emfushane ingukuthi, cha.
Ngokuqondene namazinga e-CRP , akuyona i-CRP level ngokwayo okucatshangwa ukuthi iyinkinga, kodwa kuthiwa ukuvuvukala emithanjeni ye-coronary ekhonjiswa yizinga eliphezulu le-CRP. Ngakho umbuzo wangempela wukuthi ukuvuvukala (hhayi i-CRP) kungaphathwa.
Kunobunye ubufakazi bokuthi ukutheleleka ngezinto eziphilayo okuthiwa i- Chlamydia pneumoniae kungaba yinto ebalulekile ekuthuthukiseni isifo se-coronary artery. Uma kunjalo, ama-antibiotic angase asebenze ekuqedeni ukutheleleka nokunciphisa ubungozi bokuhlaselwa kwenhliziyo (futhi, ngokusobala, ekunciphiseni amazinga e-CRP). Uma ama-antibiotic kufanele aphumelele, ukulinganisa amazinga e-CRP kungahle kube yithuluzi lokuhlola eliwusizo ukuze ukhethe iziguli ezingase zizuze kumuthi wokwelapha ama-antibiotic.
Ngaphezu kwalokho, izidakamizwa ze- statin - izidakamizwa ezisetshenziselwa ukwelapha i-cholesterol ephakeme-zingase zibe nomthelela wokuncipha ukuvuvukala emithanjeni ye-coronary. Amazinga e-CRP angahle abe yithuluzi lokuhlola lokuwusizo lapha, futhi.
I-Fibrinogen , ngokungafani ne-CRP (okucatshangwa ukuthi iyimpawu kuphela yokuvuvukala), kucatshangwa ukuthi idlala indima eqondile ekutheni i-coronary artery thrombosis. Ngakho-ke, kufanelekile uma amazinga e-fibrinogen ephakeme, ukunciphisa lawo mazinga kufanele kube umgomo wezokwelapha. Ngeshwa, azikho izindlela zokwelapha eziyaziwayo ezinciphisa amazinga e-fibrinogen.
Kungani Amazinga Wokuhlola Kubalulekile?
Yini okufanele odokotela neziguli bakwenze uma ama-CRP noma amazinga e-fibrinogen ephakanyisiwe?
Ukubuzwa ngenye indlela, uma kungekho zodwa zokwelapha ezingasetshenziswa ekuphenduleni i-CRP ephakeme noma amazinga e-fibrinogen, kungani kufanele alinganiswe?
Okwamanje, impendulo enhle kuphela yalo mbuzo yilezi: ukwazi ama-CRP namazinga e-fibrinogen angasiza ekuchazeni ngokunembile ingozi ye-coronary artery disease, ngakho-ke udokotela nesiguli banganquma ukuthi kunonya kangakanani ekuhlaseleni izinkinga ezingase zibe khona ushintshe.
Ngokwesibonelo, kokubili isiguli nodokotela bangase bangafuni ukuqala izidakamizwa ze-statin lapho amazinga e-cholesterol ephakeme kuphela. Kulokhu, i-CRP ephakeme noma amazinga e-fibrinogen angaphakamisa izikali ngenxa yokwelashwa kokuqala, kanti ama-CRP ajwayelekile noma amazinga e-fibrinogen angaphakamisa izilinganiso ukuze angabambeli ukwelapha.
Ukulinganisa eyodwa noma zombili lezi zici ezintsha zengozi, ngakho-ke, zingadlala ngqo ezinqumweni zokwelapha.
Ngokuqinisekile, ukwazi ukuthi i-CRP noma izinga le-fibrinogen liphakanyisiwe lingase libe yinsiza ekugcineni iphule emuva kwekamele - into egcina ekuphoqelele ukuthi umuntu obhemayo ayeke ukuyeka, ukuhlala emzimbeni wokuzivocavoca, noma obhejane ukushintsha kakhulu indlela yokuphila.
Kodwa kungenzeka ukuthi izilinganiso zokulinganisa ezingakwazi ukuguqulwa ngokwazo zingase zenze nje ukukhathazeka okungenakwenzeka. Kumuntu ongathembeki ngesisindo esivamile, i-cholesterol evamile, nokuphila okusebenzayo, ngokwesibonelo, kunzima ukubona ukuthi yini okungase izuze ngokwazi i-CRP ephakanyisiwe. Ngempela, kungabangela ukukhathazeka okungeke kube lula. Ngeke kube yiphutha ukwenza izilinganiso, kodwa (ezifanelana nokulinganisa izimpawu zofuzo) isiguli kufanele senziwe ngaphambi kokuba sihlole ukuthi akukho mithi ekhethekile etholakalayo. Futhi (njengama-genetic markers) anesisindo esinjalo kumlando wezokwelapha angase abe nomthelela ekuthinteni esikhathini esizayo.
Ucwaningo oluningi lwenziwa ukuze uthole izindlela zokwelapha ukuvuvukala okuthinta imithwalo ye-coronary. Uma ama-antibiotics, i-statins, noma enye yokwelapha ekugcineni iboniswa ukuthi iyasiza, ingenza umqondo omkhulu ukukala amazinga we-CRP nama-fibrinogen, ngisho nasezigulini ezingenazo ezinye izici eziyingozi.
Ukulinganisa ama-CRP namazinga e-fibrinogen angasiza emazweni amaningi futhi kungenzeka ukuthi awusizo kakhulu esikhathini esizayo. Kodwa ngaphambi kokuyala lezi zivivinyo, udokotela nesiguli kumele bakwazi ukusho kusengaphambili ukuthi imiphumela ingase ibe usizo kanjani. Ikakhulukazi ezigulini ezingenazo ezinye izinto eziyingozi, ukwenza lokhu kuhlolwa kungabangela umonakalo omkhulu kunokuhle, futhi iziguli kudingeka ziqonde ukuthi ngaphambi kokulinganisa.
Ngombhalo wokugcina, i-American Heart Association ayikhuthazi njengamanje ukuhlolwa kokujwayelekile kwe-CRP noma i-fibrinogen phakathi kwamalungu omphakathi jikelele.