Ukulimala Kwebindi Okuqhubekayo Yiyona Isizathu Esiyishumi Nesibili Sokufa E-US
I-cirrhosis yi-scartic (fibrosis) ebanzi yesibindi esabangelwa ukulimala isikhathi eside. Umonakalo ungenxa yokuvuvukala okuqhubekayo nokuqhubekayo, kanengi ukuphendula ukutheleleka ngegciwane lesandulela ngculaza njengokungenwa kwesifo sofuba noma ukudakwa ngokweqile.
Isibindi sinamandla okuzilungisa ngokwaso kepha njengoba kancane kancane sakha izicubu ezinzima, asikwazi ukusebenza kahle.
Ngokuhamba kwesikhathi, njengoba inani lokukhuphuka kwesibindi kanye nokugeleza kwegazi okuya esibindi kunciphile, imisebenzi ebalulekile yesibindi isonakele. Kwezinye izimo, lokhu kungaholela ekuhlulekeni kwesibindi ngisho nokufa.
Abantu abangaphezu kwesigidi bafa ngonyaka we-cirrhosis, kufaka phakathi ama-30,000-plus e-US Namuhla yiyona ye-12 ehola imbangela yokufa, ethinta amadoda kabili njengoba kwenza abesifazane.
Izimbangela ze-Cirrhosis
Izimbangela ezivame kakhulu ze-cirrhosis ziwukudakwa ngokweqile, isibindi se-hepatitis B , i- hepatitis C kanye nesifo esingenalo utshwala esinamafutha .
- I-Hepatitis C ibangela inani lokuxilongwa kwe-cirrhosis e-US, kanye nokuba yisikhombisa esibangela ukufakelwa kwesibindi.
- Isifo sesibindi esithinta udoti silandela emuva futhi ngokuvamile sihlobene nokuphuza ngokweqile eminyakeni eminingana (ngokwesilinganiso, ngaphezu kweziphuzo ezimbili ngosuku ngabesifazane nabangaphezu kwamathathu kwabesilisa abayishumi noma ngaphezulu).
- Nakuba i- hepatitis B engavamile e-US, ethinta abangaphansi kuka-1% wabantu, amanani angakhuphuka abe ngu-5% noma ngaphezulu emazweni amaningi asathuthuka.
- Isifo esingenalo utshwala esinamafutha ashubile ngokuvamile sihlotshaniswa nokukhuluphala, kanye nesifo sikashukela, umfutho wegazi ophakeme, ne-cholesterol ephezulu. Abantu abane-syndrome ye-metabolic, ebonakala ngobuningi obukhulu bezinwele, i-triglycerides ephezulu, izinga le-cholesterol engavamile, ukucindezela kwegazi eliphezulu, futhi ngaphezulu kunezinga elijwayelekile legazi le-glucose, lithandana kakhulu ne-cirrhosis.
Ezinye izimbangela ezingavamile kakhulu ze-cirrhosis zivinjelwe ama-bucts ebindi wesibindi ne-gallbladder, i-autoimmune hepatitis, nezifo ezifa njengezifo zika-Wilson noma i- hemochromatosis .
Izimpawu ze-Cirrhosis
Ukuqhubeka kokulimala kwesibindi kusukela ekuqaleni kwe-fibrosis kuya ku-cirrhosis kuthatha iminyaka, ngisho nangamashumi eminyaka, ukubonakalisa ngokuphawulekayo. Eminyakeni yokuqala, ngokuvamile kukhona izimpawu ezimbalwa, uma zikhona.
Uma izimpawu zivela, ngezinye izikhathi zihlukunyezwa, zinganakiwe, noma zithathwe kwezinye izimbangela ezikhona. Njengoba lesi sifo siqhubeka, noma kunjalo, izimpawu zokukhuluma zingabonakala ngokucacile futhi zibandakanya:
- ukukhathala
- ubuthakathaka
- itching
- ukulahlekelwa ukudla
- ukulahlekelwa isisindo
- isiyaluyalu
- ukululaza kalula
- i-jaundice (i-yellowing yesikhumba kanye / noma amehlo)
- isi-spider angioma (i-spider veining esikhumbeni, evame ukuzungeza izimpumputhe kanye namahlathi)
- i-edema (ukuvuvukala kwezinyawo, ama-ankle, nemilenze ngenxa yesikhumba somswakama)
- ukuvimbela esiswini kusuka phezulu (i-buildup of fluid esiswini)
Eziningi zalezi zimpawu zibangelwa yi- portal hypertension , lapho izicubu ezincane zivimbela ukugeleza kwegazi okujwayelekile esibindi.
Ukuxilongwa kwe-Cirrhosis
I-biopsy yesibindi iyindlela enembile kakhulu yokuthola i-cirrhosis nokuhlola kahle isigaba sesifo sesibindi.
Ukuhlolwa okuningi kwegazi namathuluzi okucabanga (kufaka phakathi i-ultrasound, i-CT scan kanye ne-MRI) ingasetshenziswa ukuqapha ukuqhubeka kwesifo.
I-cirrhosis ingasetshenziswa ngokujwayelekile njengokuhlawuliswa noma yokukhokhelwa . I-cirrhosis ehlawuliswayo nje isibindi esonakalisiwe esasasebenza kahle, kanti i-cirrhosis ephoqelekile isikisela ukuthi isibindi asisebenzi. Uma izinkinga zingakwazi ukulawulwa uma isibindi saphela ukusebenza, ukufakelwa kwesibindi kukhonjisiwe.
Abantu abangaba ngu-5% abane-cirrhosis bazokwakha i-hepatocellular carcinoma (HCC) , uhlobo oluvame kakhulu lomdlavuza wesibindi.
Ukwelapha i-Cirrhosis
Ukwelashwa kwe-cirrhosis kuncike kakhulu ekubambeni nesibindi saleso sifo.
Uma lesi simo siba uphawu, kuzomele kuthathwe izinyathelo eziningi ukunciphisa ukuqhuma kwesibindi, kufaka phakathi:
- Ukugwema utshwala nezinto ezingekho emthethweni (ukufuna ukwelapha izidakamizwa, uma kudingeka).
- Ukuzivocavoca ngokweqile kanye nemikhuba yokudla enamafutha aphansi.
- Gwema i-shellfish eluhlaza, ene-bacterium engabangela ukutheleleka okunzulu kubantu abanezifo ezinamandla zesibindi.
- Imishanguzo ye-hepatitis A ne- hepatitis B , kanye nokuhlolwa kwesifo sofuba C.
- Ukwelashwa kwe-antiviral ye-hepatitis B ne-hepatitis C.
- Ukwelapha ezinye izimbangela ze-cirrhosis (isibonelo, i- ursodiol ye-bile duct blockages).
Imithombo:
I-National Institute of Diabetes kanye ne-Digestive and Kidney Diseases. "I-cirrhosis." I-Bethesda, e-Maryland; kufinyelelwe ngoJanuwari 1, 2015.
Amasevisi Okulawula Nokuvimbela Izifo (CDC). "Ukufa: Idatha Yokugcina ka-2010." Umbiko we-National Vital Statistics. May 8, 2013; 61 (4): 1-118.