Ubuhlungu obuvame ukuhamba nge- infarction ye-myocardial (ukuhlaselwa kwenhliziyo) kubangelwa ukuvinjelwa okunzima komunye wemithanguzo ye-coronary, ngokuvamile ngenxa ye- coronary artery disease (CAD) . Lokhu kuvinjelwa okungazelelwe kungumphumela weplate e-rupture atherosclerotic odongeni lomthambo.
Lapho umthambo we-coronary uvinjelwa, imisipha yenhliziyo ehlinzekwa yilo mshini ngokushesha iba ischemic enzima (i-oksijini ilambile), futhi uma i-ischemia iphikelela ama-cell muscle cell aqala ukufa.
Ukufa kwesisu senhliziyo yilokho okuchaza ukuhlasela kwenhliziyo.
Ukuhlasela kwenhliziyo kuvame ukuveza izimpawu ezibalulekile. Ubuhlungu besifuba, noma ukuhlukumeza kwesifuba, ngokuvamile kuyingxenye evelele yalezi zimpawu.
Izinhlamvu zobuhlungu
Ubuhlungu obuhambisana nesifo senhliziyo ngokuvamile kufana ne- angina yesi-classic, kodwa kubhekwa ukuthi kube nzima kakhulu futhi buhlala isikhathi eside. Ubuhlungu obukhulu buchazwa njengengcindezi efana nokucindezela, ukuchoboza, noma ubuhlungu obunzima. Ubuhlungu buvame ukuqhuma emhlathini, emahlombe noma ezandleni.
Kodwa-ke, ubuhlungu bezinhlungu zenhliziyo kaningi buba "obuthakathaka." Ngezinye izikhathi akubandakanyi esifubeni, kepha kunalokho kungase kube phakathi, ngemuva kokubili, noma esiswini. Ngaphezu kwalokho, kungase kungabonakali isisulu ngokuthi "ubuhlungu" nhlobo, kodwa kunzima.
Ngaphezu kokubuhlungu (noma ukungakhululeki) abantu abahlaselwa yinhliziyo bayokwazi ukubona i-dyspnea (ukuphefumula), kanye nesicashu, ukujuluka, isizungu, ubuthakathaka, noma ukukhathala okungaqondakali.
Ngokungafani ne-angina, izimpawu zivame ukuphikelela, futhi ngokuvamile zakha okungenani imizuzu engu-15 noma engu-20 yokuqala.
Lezi zimpawu zivame ukukhula ngokuzumayo - kodwa lokhu akuwona umthetho. Izimpawu zesifo senhliziyo zingakha kancane, ngaphezu kwamahora noma ngisho nezinsuku.
Izimpawu zesifo senhliziyo ngokuvamile zihambisana nokuzwa ukwesaba okukhulu - okuchazwa ngokweqile ngokuthi "umuzwa wengozi ezayo."
Okufanele ngikwenze
Noma ubani obhekene nezimpawu ezifana nesifo senhliziyo sidinga usizo lwezokwelapha ngokushesha. Uma kuyisifo senhliziyo, imizuzu ingenza umehluko phakathi kokuphila okude nokujabulisayo, noma ukukhubazeka okuhlala njalo noma ukufa. Shayela u-9-1-1.
Uma ufika emnyangweni ophuthumayo, ukuhlolwa kukadokotela kuzohlanganisa umlando wezokwelapha ngokucophelela nokuhlolwa ngokomzimba, okuvame ukukhomba ukuxilongwa, kanye ne- ECG , evame ukuthola ukuxilongwa. Ama-enzyme e-cardiac (amaprotheni enhliziyo awela egazini lapho amangqamuzana e-cardiac efa) alinganiswa ekuhlolweni kwegazi ukuqinisekisa ukuxilongwa. Ukuhlolwa kokuhlaselwa kwenhliziyo kufanele kwenzeke ngokushesha futhi ngokusheshisa, ngoba ukwelashwa kokuqala kanye nolaka kungalondoloza izicubu zenhliziyo futhi kuthuthukise kakhulu amathuba okuphila kwakho okukhubazekile.
Imithombo:
U-O'Gara PT, uKushner FG, u-Ascheim DD, et al. 2013 ACCF / AHA isiqondiso sokuphathwa kwe-ST-elevation infarction ye-ST-elevation infarction: umbiko we-American College of Cardiology Foundation / American Heart Association Task Force on Guidelines Practice. Ukujikeleza 2013; 127: e362.
Svavarsdottir, AE, Jonasson, MR, Gudmundsson, GH, Fjeldsted, K. Ubuhlungu be-Chest emisebenzini yomndeni. Ukuxilongwa kanye nomphumela wesikhathi eside esimweni somphakathi. Ingabe i-Fam Physician; 42: 1122.