Singasondeza Kanjani Kuphi Ukwelashwa Okusebenzayo Kwe-HIV?

Ukuthola Okubalulekile Kuvula Indlela Yokudlulisa Isikhathi Eside

Ukwelashwa okusebenzayo kuyimbono esuselwa ebufakazini lapho i-HIV ingagcinwa khona ngaphandle kokusebenzisa imithi engapheli. Ngokungafani nomgomo wokugoma , lapho i-HIV izoqedwa ngokuphelele emzimbeni, ukwelashwa okusebenzayo kusebenza kakhulu emigqeni yokukhululwa lapho igciwane lingakwazi ukubangela ukugula ngisho noma ngabe isifo segciwane sisaphila.

Kube nomdlandla omkhulu kanye nokuphikisana okuningi okuzungeze ithemba lomuthi osebenzayo. UFrançoise Barré-Sinoussi , ohlangene ngokuthola i-HIV, wathi ngo-2013 ukuthi ukholelwa ngokugcwele ukuthi ukwelashwa okunjalo kungatholakaliswa "phakathi neminyaka engu-30 ezayo." Ngokuphambene nalokho, uRobert Gallo (ophinde wabizwa ngokuthi uthola i-HIV) ubheka umqondo ophutha futhi ukholelwa ukuthi izingxenye ze-theory "azikwazi ukusebenza."

Yeka indlela ukwelashwa okusebenzayo okusebenza ngayo

Enye yezinselelo ezinkulu ezibhekene nabacwaningi kuye kwaba ngamaseli kanye nezicubu zomzimba (ezibizwa ngokuthi izibhamu ezingavamile ) lapho i-HIV ingakwazi ukuphikelela ngisho nokubhekana nokucindezelwa okuphelele kwegciwane. Efihliwe ngaphakathi kwalezi zikhungo zamagesi kukhona i-genetic code ye-HIV, okuyinto amasosha omzimba akakwazi ukuwabona.

Njengoba igciwane lingenzi lutho ngokuphindaphindiwe-kodwa kunalokho liqhutshwa ngokunyathelisa njengoba iseli lendawo liphikisana-ikakhulukazi lingathinti izidakamizwa ezidambisigciwane (njengoba umsebenzi we-antiretrovirals ngokuphazamisa isigaba emjikelezweni wokuphila wegciwane, hhayi umphathi).

Kunezibonelo eziningana ezihlolwe ukubhekana nalokhu:

Ubufakazi Ekusekeleni Ukwelashwa Okusebenzayo

Ngenkathi ucwaningo olwenziwe ngomuthi osebenzayo lube setafuleni iminyaka eminingana, izenzakalo ezintathu eziqondile zanikeza ubufakazi obuyisisekelo-bombono.

Omkhulu phakathi kwabo yisiguli esisodwa esicatshangelwa ukuthi "siphulukiswa" se-HIV ngonyaka ka-2009. UThimothy Brown (umguli waseBerlin) wayenomuntu ophila negciwane lesandulela ngculaza ehlala eBerlin, owanikezwa ukuthambisa umnkantsha wamathambo ukuze aphule i-leukemia yakhe. Odokotela bakhetha umnikeli wesisindo se-stem ngamakhophi amabili okuguqulwa kwezakhi zofuzo okuthiwa i-CCR5-delta-32, eyaziwa ukulwa negciwane lesandulela ngculazi kubantu abaningi abangavamile .

Ukuhlola okujwayelekile kwenziwa ngokushesha ngemva kokuba ukufakelwa kwembulani ukuthi ama-antibodies e-HIV egazini likaBrown ayehlile emazingeni anjengokuphakamisa ukwedluliswa kwegciwane. Ama-biopsies alandelayo aqinisekisile ubufakazi be-HIV kunoma yisiphi isisindo sikaBrown, esisekela izimangalo ukuthi lo muntu wayephilile ngempela. Ngenkathi ingozi yokufa ibhekwa kakhulu ukuba ihlolwe ukuguqulwa kwamasongo njengendlela yokunciphisa, icala linikeze okungenani ubufakazi bokuthi ukwelashwa kungenzeka, empeleni kungenzeka.

Phakathi naleso sikhathi, abanye ososayensi baye baphenya amakhemikhali okuhlola ayenamandla okuhlanza igciwane lesandulela ngculazi emagodini abo okugcina.

Olunye lwezifundo zakuqala, oluqhutshwa eNyuvesi yaseNorth Carolina ngo-2009 lwabonisa ukuthi isigaba sezidakamizwa okuthiwa i- histone deacetylase (HDAC) inhibitors singenza kabusha i-HIV engavamile kumazinga okudakamizwa abhekwe ephephile futhi ebekezeleleka.

Nakuba izifundo ezalandela ziphakamise ukuthi ukusetshenziswa kwe-agent eyodwa ye-HDAC kungase kuhlinzeke kuphela ukuvuselelwa okuyingxenye, kunobunye ubufakazi bokuphakamisa ukuthi ukuhlanganiswa kwe-HDAC noma amakilasi amasha wemithi elwa nomdlavuza (ebizwa ngokuthi i-ingenol compounds) ingase igcwalise ngokuphelele i-HIV engavamile kusuka ekufihliwe kwayo izibhamu.

I-Path Phambili

Njengokwethenjelwa njengoba yonke ucwaningo lungabonakala, baphakamisa imibuzo eminingi njengoba bephendula. Omkhulu phakathi kwabo:

Ngenkathi sibonakala siyindlela efanele, kubalulekile ukubuka ucwaningo ngokulindela okuhle. Ngisho njengoba ososayensi beqhubeka bevula izimfihlakalo ezizungeze i-HIV, akukho nhlobo yalezi zintuthuko ezibonisa ukuthi imithetho ephathelene nokuvimbela nokwelashwa kwe-HIV ishintshile.

Uma kukhona, kunikezwa ubufakazi bokuthi ukutholakala kokuqala nokungenelela kuyisisekelo sokwelashwa, okubalulekile ukuhlala uqaphile, mhlawumbe, kubaluleke kunanini ngaphambili.

Imithombo:

> Hütter, G .; Manjeak, D .; Mossner, M .; et al. "Ukulawula Kwesikhathi Eside Ne-HIV nge-CCR5 Delta32 / Delta32 Isitembu Sokufakelwa Kwamaselula." I-New England Journal of Medicine. Ngo-February 12, 2009; 360: 692-698.

> Archim, N .; U-Espeseth, A .; UMargolis, D .; et al. "Ukuboniswa kwe-HIV elandelwayo yenziwa yi-Potential HDAC Inhibitor Suberoylanilide Hydroxamic Acid." I-AID Research of Retroviruses Abantu. Februwari 2009; 25 (2): 207-212.

> Sáez-Cirión, A .; Bacchus, C .; I-Hocqueloux, i-L .; et al. "Abaphathi be-Post-Treatment HIV-1 abane-Remote Virological Remission emva kokuphazanyiswa kwe-Antiretroviral Early Early Treatment ANRS VISCONTI." I-PLoS Pathology. Ngo-Mashi 14, 2013; 0 (3): e1003211.

> Jiang, G .; I-Mendes, E .; Kaiser, P .; et al. "Ukuvuselelwa kwe-Synergistic ye-Latent Expression ye-Ingenol-3-Angelate, i-PEP005, ekhonjisiwe i-NF-kB Ukubonakaliswa kokuhlanganiswa ne-JQ1 eyenziwe nge-P-TEFb Ukuqalisa." I-PLoS Pathogens. Julayi 30 2015; I-DOI: 10.1371 / iphephandaba.ppat.1005066.